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RIP3: a molecular switch for necrosis and inflammation

The receptor-interacting protein kinase 3 (RIP3/RIPK3) has emerged as a critical regulator of programmed necrosis/necroptosis, an inflammatory form of cell death with important functions in pathogen-induced and sterile inflammation. RIP3 activation is tightly regulated by phosphorylation, ubiquitina...

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Autors principals: Moriwaki, Kenta, Chan, Francis Ka-Ming
Format: Artigo
Idioma:Inglês
Publicat: Cold Spring Harbor Laboratory Press 2013
Matèries:
Accés en línia:https://ncbi.nlm.nih.gov/pmc/articles/PMC3744722/
https://ncbi.nlm.nih.gov/pubmed/23913919
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1101/gad.223321.113
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