Ceftriaxone Treatment after Traumatic Brain Injury Restores Expression of the Glutamate Transporter, GLT-1, Reduces Regional Gliosis, and Reduces Post-Traumatic Seizures in the Rat

Excessive extracellular glutamate after traumatic brain injury (TBI) contributes to excitotoxic cell death and likely to post-traumatic epilepsy. Glutamate transport is the only known mechanism of extracellular glutamate clearance, and glutamate transporter 1 (GLT-1) is the major glutamate transport...

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Bibliografiska uppgifter
Huvudupphovsmän: Goodrich, Grant S., Kabakov, Anatoli Y., Hameed, Mustafa Q., Dhamne, Sameer C., Rosenberg, Paul A., Rotenberg, Alexander
Materialtyp: Artigo
Språk:Inglês
Publicerad: Mary Ann Liebert, Inc. 2013
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Original Articles
Länkar:https://ncbi.nlm.nih.gov/pmc/articles/PMC3741415/
https://ncbi.nlm.nih.gov/pubmed/23510201
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1089/neu.2012.2712
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