Ceftriaxone Treatment after Traumatic Brain Injury Restores Expression of the Glutamate Transporter, GLT-1, Reduces Regional Gliosis, and Reduces Post-Traumatic Seizures in the Rat

Samankaltaisia teoksia

• Evidence for change in current flux coupling of GLT1 at high glutamate concentrations in primary forebrain neurons and GLT1a expressing COS7 cells
  Tekijä: Kabakov, Anatoli Y., et al.
  Julkaistu: (2009)
• GLT-1 Transport Stoichiometry Is Constant at Low and High Glutamate Concentrations when Chloride Is Substituted by Gluconate
  Tekijä: Kabakov, Anatoli Y., et al.
  Julkaistu: (2015)
• Conditional Deletion of the Glutamate Transporter GLT-1 Reveals That Astrocytic GLT-1 Protects against Fatal Epilepsy While Neuronal GLT-1 Contributes Significantly to Glutamate Uptake into Synaptosomes
  Tekijä: Petr, Geraldine T., et al.
  Julkaistu: (2015)
• Ceftriaxone Treatment Preserves Cortical Inhibitory Interneuron Function via Transient Salvage of GLT-1 in a Rat Traumatic Brain Injury Model
  Tekijä: Hameed, Mustafa Q, et al.
  Julkaistu: (2019)
• Characterization of the Visceral Antinociceptive Effect of Glial Glutamate Transporter GLT-1 Upregulation by Ceftriaxone
  Tekijä: Roman, K., et al.
  Julkaistu: (2012)