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SMAD1-deficiency in either endothelial or smooth muscle cells can predispose mice to pulmonary hypertension
A deficiency in bone morphogenetic protein receptor type 2 (BMPR2) signaling is a central contributor in the pathogenesis of pulmonary arterial hypertension (PAH). We have recently shown that endothelial-specific Bmpr2-deletion by a novel L1Cre line resulted in pulmonary hypertension. SMAD1 is one o...
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| Hlavní autoři: | , , , , , , , , |
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| Médium: | Artigo |
| Jazyk: | Inglês |
| Vydáno: |
2013
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| Témata: | |
| On-line přístup: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3740527/ https://ncbi.nlm.nih.gov/pubmed/23478097 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1161/HYPERTENSIONAHA.111.199158 |
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