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Presynaptic homeostasis at CNS nerve terminals compensates for lack of a key Ca(2+) entry pathway
At central synapses, P/Q-type Ca(2+) channels normally provide a critical Ca(2+) entry pathway for neurotransmission. Nevertheless, we found that nerve terminals lacking α(1A) (Ca(V)2.1), the pore-forming subunit of P/Q-type channels, displayed a remarkable preservation of synaptic function. Two con...
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| Main Authors: | , , , , , , , , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
National Academy of Sciences
2004
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC373510/ https://ncbi.nlm.nih.gov/pubmed/14990796 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1073/pnas.0308188100 |
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