Presynaptic homeostasis at CNS nerve terminals compensates for lack of a key Ca(2+) entry pathway

At central synapses, P/Q-type Ca(2+) channels normally provide a critical Ca(2+) entry pathway for neurotransmission. Nevertheless, we found that nerve terminals lacking α(1A) (Ca(V)2.1), the pore-forming subunit of P/Q-type channels, displayed a remarkable preservation of synaptic function. Two con...

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Main Authors: Piedras-Rentería, Erika S., Pyle, Jason L., Diehn, Max, Glickfeld, Lindsey L., Harata, Nobutoshi C., Cao, Yuqing, Kavalali, Ege T., Brown, Patrick O., Tsien, Richard W.
Formáid: Artigo
Teanga:Inglês
Foilsithe: National Academy of Sciences 2004
Ábhair:
Biological Sciences
Rochtain Ar Líne:https://ncbi.nlm.nih.gov/pmc/articles/PMC373510/
https://ncbi.nlm.nih.gov/pubmed/14990796
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1073/pnas.0308188100
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