Modulation of L-type Ca(2+) current but not activation of Ca(2+) release by the gamma(1) subunit of the dihydropyridine receptor of skeletal muscle

Documenti analoghi

• Truncation of the Carboxyl Terminus of the DihydropyridineReceptor β1a Subunit Promotes Ca(2+) Dependent Excitation-Contraction Coupling in Skeletal Myotubes
  di: Sheridan, David C., et al.
  Pubblicazione: (2003)
• Reduced Ca2+ current, charge movement, and absence of Ca2+ transients in skeletal muscle deficient in dihydropyridine receptor beta 1 subunit.
  di: Strube, C, et al.
  Pubblicazione: (1996)
• Molecular origin of the L-type Ca2+ current of skeletal muscle myotubes selectively deficient in dihydropyridine receptor beta1a subunit.
  di: Strube, C, et al.
  Pubblicazione: (1998)
• Functional analysis of a frame-shift mutant of the dihydropyridine receptor pore subunit (α(1S)) expressing two complementary protein fragments
  di: Ahern, Chris A, et al.
  Pubblicazione: (2001)
• Ca(2+) Current and Charge Movements in Skeletal Myotubes Promoted by the β-Subunit of the Dihydropyridine Receptor in the Absence of Ryanodine Receptor Type 1
  di: Ahern, Chris A., et al.
  Pubblicazione: (2003)