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USP18 inhibits NF-κB and NFAT activation during Th17 differentiation by deubiquitinating the TAK1–TAB1 complex

Reversible ubiquitin modification of cell signaling molecules has emerged as a critical mechanism by which cells respond to extracellular stimuli. Although ubiquitination of TGF-β–activated kinase 1 (TAK1) is critical for NF-κB activation in T cells, the regulation of its deubiquitination is unclear...

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Bibliografiske detaljer
Main Authors: Liu, Xikui, Li, Hongxiu, Zhong, Bo, Blonska, Marzenna, Gorjestani, Sara, Yan, Ming, Tian, Qiang, Zhang, Dong-Er, Lin, Xin, Dong, Chen
Format: Artigo
Sprog:Inglês
Udgivet: The Rockefeller University Press 2013
Fag:
Online adgang:https://ncbi.nlm.nih.gov/pmc/articles/PMC3727316/
https://ncbi.nlm.nih.gov/pubmed/23825189
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1084/jem.20122327
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