Corticosteroids inhibit complement-induced granulocyte aggregation. A possible mechanism for their efficacy in shock states.
Granulocyte (PMN) aggregation and embolization may underlie complement (C)-mediated organ dysfunction in such syndromes as hemodialysis neutropenia and Purtscher's ischem;c retinopathy. Because of clinical and pathologic parallels, we have further suggested a role for this phenomenon in the genesis...
Furkejuvvon:
| Publikašuvnnas: | J Clin Invest |
|---|---|
| Váldodahkkit: | , , , |
| Materiálatiipa: | Artigo |
| Giella: | Inglês |
| Almmustuhtton: |
American Society for Clinical Investigation
1979
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| Fáttát: | |
| Liŋkkat: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC372017/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/438338/ https://ncbi.nlm.nih.govhttps://doi.org/10.1172/JCI109365 |
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