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Corticosteroids inhibit complement-induced granulocyte aggregation. A possible mechanism for their efficacy in shock states.

Granulocyte (PMN) aggregation and embolization may underlie complement (C)-mediated organ dysfunction in such syndromes as hemodialysis neutropenia and Purtscher's ischem;c retinopathy. Because of clinical and pathologic parallels, we have further suggested a role for this phenomenon in the genesis...

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Bibliográfalaš dieđut
Publikašuvnnas:J Clin Invest
Váldodahkkit: Hammerschmidt, D E, White, J G, Craddock, P R, Jacob, H S
Materiálatiipa: Artigo
Giella:Inglês
Almmustuhtton: American Society for Clinical Investigation 1979
Fáttát:
Liŋkkat:https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC372017/
https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/438338/
https://ncbi.nlm.nih.govhttps://doi.org/10.1172/JCI109365
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