Načítá se...

Blockade of KCa3.1 Ameliorates Renal Fibrosis Through the TGF-β1/Smad Pathway in Diabetic Mice

The Ca(2+)-activated K(+) channel KCa3.1 mediates cellular signaling processes associated with dysfunction of vasculature. However, the role of KCa3.1 in diabetic nephropathy is unknown. We sought to assess whether KCa3.1 mediates the development of renal fibrosis in two animal models of diabetic ne...

Celý popis

Uloženo v:
Podrobná bibliografie
Hlavní autoři: Huang, Chunling, Shen, Sylvie, Ma, Qing, Chen, Jason, Gill, Anthony, Pollock, Carol A., Chen, Xin-Ming
Médium: Artigo
Jazyk:Inglês
Vydáno: American Diabetes Association 2013
Témata:
On-line přístup:https://ncbi.nlm.nih.gov/pmc/articles/PMC3717839/
https://ncbi.nlm.nih.gov/pubmed/23656889
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.2337/db13-0135
Tagy: Přidat tag
Žádné tagy, Buďte první, kdo otaguje tento záznam!