Blockade of KCa3.1 Ameliorates Renal Fibrosis Through the TGF-β1/Smad Pathway in Diabetic Mice

The Ca(2+)-activated K(+) channel KCa3.1 mediates cellular signaling processes associated with dysfunction of vasculature. However, the role of KCa3.1 in diabetic nephropathy is unknown. We sought to assess whether KCa3.1 mediates the development of renal fibrosis in two animal models of diabetic ne...

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Autors principals: Huang, Chunling, Shen, Sylvie, Ma, Qing, Chen, Jason, Gill, Anthony, Pollock, Carol A., Chen, Xin-Ming
Format: Artigo
Idioma:Inglês
Publicat: American Diabetes Association 2013
Matèries:
Original Research
Accés en línia:https://ncbi.nlm.nih.gov/pmc/articles/PMC3717839/
https://ncbi.nlm.nih.gov/pubmed/23656889
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.2337/db13-0135
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