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Reinterpreting the Action of ATP Analogs on K(ATP) Channels
Neuroendocrine-type K(ATP) channels, (SUR1/Kir6.2)(4), couple the transmembrane flux of K(+), and thus membrane potential, with cellular metabolism in various cell types including insulin-secreting β-cells. Mutant channels with reduced activity are a cause of congenital hyperinsulinism, whereas hype...
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| Huvudupphovsmän: | , , , |
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| Materialtyp: | Artigo |
| Språk: | Inglês |
| Publicerad: |
American Society for Biochemistry and Molecular Biology
2013
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| Ämnen: | |
| Länkar: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3696665/ https://ncbi.nlm.nih.gov/pubmed/23665564 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1074/jbc.M113.476887 |
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