Brain-Derived Neurotrophic Factor-Dependent Synaptic Plasticity Is Suppressed by Interleukin-1β via p38 Mitogen-Activated Protein Kinase

Evolving evidence suggests that brain inflammation and the buildup of proinflammatory cytokine increases the risk for cognitive decline and cognitive dysfunction. Interleukin-1β (IL-1β), acting via poorly understood mechanisms, appears to be a key cytokine in causing these deleterious effects along...

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Bibliographic Details
Main Authors:	Tong, Liqi, Prieto, G. Aleph, Kramár, Enikö A., Smith, Erica D., Cribbs, David H., Lynch, Gary, Cotman, Carl W.
Format:	Artigo
Language:	Inglês
Published:	Society for Neuroscience 2012
Subjects:	Articles
Online Access:	https://ncbi.nlm.nih.gov/pmc/articles/PMC3687587/ https://ncbi.nlm.nih.gov/pubmed/23223292 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.1253-12.2012
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Brain-Derived Neurotrophic Factor-Dependent Synaptic Plasticity Is Suppressed by Interleukin-1β via p38 Mitogen-Activated Protein Kinase

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