Excessive bone formation in a mouse model of ankylosing spondylitis is associated with decreases in Wnt pathway inhibitors

INTRODUCTION: Ankylosing spondylitis (AS) is unique in its pathology where inflammation commences at the entheses before progressing to an osteoproliferative phenotype generating excessive bone formation that can result in joint fusion. The underlying mechanisms of this progression are poorly unders...

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Bibliografiske detaljer
Main Authors:	Haynes, Katelin R, Pettit, Allison R, Duan, Ran, Tseng, Hsu-Wen, Glant, Tibor T, Brown, Matthew A, Thomas, Gethin P
Format:	Artigo
Sprog:	Inglês
Udgivet:	BioMed Central 2012
Fag:	Research Article
Online adgang:	https://ncbi.nlm.nih.gov/pmc/articles/PMC3674607/ https://ncbi.nlm.nih.gov/pubmed/23171658 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1186/ar4096
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Excessive bone formation in a mouse model of ankylosing spondylitis is associated with decreases in Wnt pathway inhibitors

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