Cardiac-specific mindin overexpression attenuates cardiac hypertrophy via blocking AKT/GSK3β and TGF-β1–Smad signalling

AIMS: Mindin is a secreted extracellular matrix protein, an integrin ligand, and an angiogenesis inhibitor, other examples of which are all key players in the progression of cardiac hypertrophy. However, its function during cardiac hypertrophy remains unclear. This study was aimed to identify the ef...

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Dades bibliogràfiques
Autors principals: Yan, Ling, Wei, Xiang, Tang, Qi-Zhu, Feng, Jinghua, Zhang, Yan, Liu, Chen, Bian, Zhou-Yan, Zhang, Lian-Feng, Chen, Manyin, Bai, Xue, Wang, Ai-Bing, Fassett, John, Chen, Yingjie, He, You-Wen, Yang, Qinglin, Liu, Peter P., Li, Hongliang
Format: Artigo
Idioma:Inglês
Publicat: Oxford University Press 2011
Matèries:
Original Articles
Accés en línia:https://ncbi.nlm.nih.gov/pmc/articles/PMC3657538/
https://ncbi.nlm.nih.gov/pubmed/21632881
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1093/cvr/cvr159
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