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Dysregulated Autophagy Contributes to Podocyte Damage in Fabry’s Disease
Fabry’s disease results from an inborn error of glycosphingolipid metabolism that is due to deficiency of the lysosomal hydrolase α-galactosidase A. This X-linked defect results in the accumulation of enzyme substrates with terminally α-glycosidically bound galactose, mainly the neutral glycosphingo...
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| Κύριοι συγγραφείς: | , , , , , , , , , , , |
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| Μορφή: | Artigo |
| Γλώσσα: | Inglês |
| Έκδοση: |
Public Library of Science
2013
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| Θέματα: | |
| Διαθέσιμο Online: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3656911/ https://ncbi.nlm.nih.gov/pubmed/23691056 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1371/journal.pone.0063506 |
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