Acidosis Activation of the Proton-Sensing GPR4 Receptor Stimulates Vascular Endothelial Cell Inflammatory Responses Revealed by Transcriptome Analysis

Lignende værker

• Activation of GPR4 by Acidosis Increases Endothelial Cell Adhesion through the cAMP/Epac Pathway
  af: Chen, Aishe, et al.
  Udgivet: (2011)
• Acidosis Activates Endoplasmic Reticulum Stress Pathways through GPR4 in Human Vascular Endothelial Cells
  af: Dong, Lixue, et al.
  Udgivet: (2017)
• The Proton-Sensing GPR4 Receptor Regulates Paracellular Gap Formation and Permeability of Vascular Endothelial Cells
  af: Elizabeth A. Krewson, et al.
  Udgivet: (2020-02-01)
• The Proton-Sensing GPR4 Receptor Regulates Paracellular Gap Formation and Permeability of Vascular Endothelial Cells
  af: Krewson, Elizabeth A., et al.
  Udgivet: (2020)
• Acidosis Decreases c-Myc Oncogene Expression in Human Lymphoma Cells: A Role for the Proton-Sensing G Protein-Coupled Receptor TDAG8
  af: Li, Zhigang, et al.
  Udgivet: (2013)