Ligand and protein kinase C downmodulate the colony-stimulating factor 1 receptor by independent mechanisms.
The turnover of the colony-stimulating factor 1 receptor (CSF-1R), the c-fms proto-oncogene product, is accelerated by ligand binding or by activators of protein kinase C (PKC), such as the phorbol ester 12-O-tetradecanoylphorbol-13-acetate (TPA). The mechanisms of ligand- and TPA-induced downmodula...
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| 出版年: | Mol Cell Biol |
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| 主要な著者: | , , |
| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
Taylor & Francis
1989
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC362755/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/2528680/ https://ncbi.nlm.nih.govhttps://doi.org/10.1128/mcb.9.7.2890-2896.1989 |
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