Transforming mutant v-mos protein kinases that are deficient in in vitro autophosphorylation.
We investigated the importance of specific serine residues for autophosphorylation and transformation by serine-threonine protein kinase p37mos. When either serine 326 or 358 was replaced with alanine, the resulting mutant protein retained the ability to transform NIH 3T3 cells but failed to autopho...
保存先:
| 出版年: | Mol Cell Biol |
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| 主要な著者: | , |
| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
Taylor & Francis
1989
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC362478/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/2528689/ https://ncbi.nlm.nih.govhttps://doi.org/10.1128/mcb.9.9.4087-4090.1989 |
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