Transforming mutant v-mos protein kinases that are deficient in in vitro autophosphorylation.
We investigated the importance of specific serine residues for autophosphorylation and transformation by serine-threonine protein kinase p37mos. When either serine 326 or 358 was replaced with alanine, the resulting mutant protein retained the ability to transform NIH 3T3 cells but failed to autopho...
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| Publicado no: | Mol Cell Biol |
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| Principais autores: | , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
Taylor & Francis
1989
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC362478/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/2528689/ https://ncbi.nlm.nih.govhttps://doi.org/10.1128/mcb.9.9.4087-4090.1989 |
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