Transcomplementation by a Truncation Mutant of Cystic Fibrosis Transmembrane Conductance Regulator (CFTR) Enhances ΔF508 Processing through a Biomolecular Interaction

Lignende værker

• Cystic Fibrosis Transmembrane Regulator Missing the First Four Transmembrane Segments Increases Wild Type and ΔF508 Processing
  af: Cebotaru, Liudmila, et al.
  Udgivet: (2008)
• Syntaxin 6 and CAL Mediate the Degradation of the Cystic Fibrosis Transmembrane Conductance Regulator
  af: Cheng, Jie, et al.
  Udgivet: (2010)
• Syntaxin 8 and the Endoplasmic Reticulum Processing of ΔF508-CFTR
  af: Sabirzhanova, Inna, et al.
  Udgivet: (2018)
• Rescuing cystic fibrosis transmembrane conductance regulator (CFTR)-processing mutants by transcomplementation
  af: Cormet-Boyaka, Estelle, et al.
  Udgivet: (2004)
• The CFTR-Associated Ligand arrests the Trafficking of the Mutant ΔF508 CFTR Channel in the ER contributing to Cystic Fibrosis
  af: Bergbower, E.A.S., et al.
  Udgivet: (2018)