The tumor promoter 12-O-tetradecanoylphorbol-13-acetate and the ras oncogene modulate expression and phosphorylation of gap junction proteins.

Podobne zapisy

• Lovastatin selectively inhibits ras activation of the 12-O-tetradecanoylphorbol-13-acetate response element in mammalian cells.
  od: Defeo-Jones, D, i wsp.
  Wydane: (1991)
• RasGRP1 Is Essential for Ras Activation by the Tumor Promoter 12-O-Tetradecanoylphorbol-13-acetate in Epidermal Keratinocytes
  od: Sharma, Amrish, i wsp.
  Wydane: (2010)
• 12-O-Tetradecanoylphorbol-13-Acetate Induces Keratin 8 Phosphorylation and Reorganization via Expression of Transglutaminase-2
  od: Lee, Eun Ji, i wsp.
  Wydane: (2014)
• Two overlapping sequence motifs within the polyomavirus enhancer are independently the targets of stimulation by both the tumor promoter 12-O-tetradecanoylphorbol-13-acetate and the Ha-ras oncogene.
  od: Yamaguchi, Y, i wsp.
  Wydane: (1989)
• Switch in gap junction protein expression is associated with selective changes in junctional permeability during keratinocyte differentiation.
  od: Brissette, J L, i wsp.
  Wydane: (1994)