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Prion Protein-mediated Toxicity of Amyloid-β Oligomers Requires Lipid Rafts and the Transmembrane LRP1

Soluble oligomers of the amyloid-β (Aβ) peptide cause neurotoxicity, synaptic dysfunction, and memory impairments that underlie Alzheimer disease (AD). The cellular prion protein (PrP(C)) was recently identified as a high affinity neuronal receptor for Aβ oligomers. We report that fibrillar Aβ oligo...

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Bibliografiset tiedot
Päätekijät: Rushworth, Jo V., Griffiths, Heledd H., Watt, Nicole T., Hooper, Nigel M.
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: American Society for Biochemistry and Molecular Biology 2013
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Linkit:https://ncbi.nlm.nih.gov/pmc/articles/PMC3610967/
https://ncbi.nlm.nih.gov/pubmed/23386614
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1074/jbc.M112.400358
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