Central role of the exchange factor GEF-H1 in TNF-α–induced sequential activation of Rac, ADAM17/TACE, and RhoA in tubular epithelial cells

Lignende værker

• Extracellular signal regulated kinase and GEF-H1 mediate depolarization-induced Rho activation and paracellular permeability increase
  af: Waheed, Faiza, et al.
  Udgivet: (2010)
• GEF-H1 Mediates Tumor Necrosis Factor-α-induced Rho Activation and Myosin Phosphorylation: ROLE IN THE REGULATION OF TUBULAR PARACELLULAR PERMEABILITY
  af: Kakiashvili, Eli, et al.
  Udgivet: (2009)
• The Epidermal Growth Factor Receptor Mediates Tumor Necrosis Factor-α-induced Activation of the ERK/GEF-H1/RhoA Pathway in Tubular Epithelium
  af: Kakiashvili, Eli, et al.
  Udgivet: (2011)
• Claudin-2 suppresses GEF-H1, RHOA, and MRTF, thereby impacting proliferation and profibrotic phenotype of tubular cells
  af: Dan, Qinghong, et al.
  Udgivet: (2019)
• Tumor necrosis factor-α induces a biphasic change in claudin-2 expression in tubular epithelial cells: role in barrier functions
  af: Amoozadeh, Yasaman, et al.
  Udgivet: (2015)