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Rapamycin Reverses Status Epilepticus-Induced Memory Deficits and Dendritic Damage
Cognitive impairments are prominent sequelae of prolonged continuous seizures (status epilepticus; SE) in humans and animal models. While often associated with dendritic injury, the underlying mechanisms remain elusive. The mammalian target of rapamycin complex 1 (mTORC1) pathway is hyperactivated f...
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| Hlavní autoři: | , , , , , , |
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| Médium: | Artigo |
| Jazyk: | Inglês |
| Vydáno: |
Public Library of Science
2013
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| Témata: | |
| On-line přístup: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3594232/ https://ncbi.nlm.nih.gov/pubmed/23536771 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1371/journal.pone.0057808 |
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