Transactivation of the human p53 tumor suppressor gene by c-Myc/Max contributes to elevated mutant p53 expression in some tumors.

Elevated levels of mutant forms of the p53 tumor suppressor are a hallmark of many transformed cells. Multiple mechanisms such as increased stability of the protein and increased transcription of the gene can account for elevated p53 expression. Recent findings indicate that c-Myc/Max heterodimers c...

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Vydáno v:	Mol Cell Biol
Hlavní autoři:	Roy, B, Beamon, J, Balint, E, Reisman, D
Médium:	Artigo
Jazyk:	Inglês
Vydáno:	Taylor & Francis 1994
Témata:	Research Article
On-line přístup:	https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC359320/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/7969121/ https://ncbi.nlm.nih.govhttps://doi.org/10.1128/mcb.14.12.7805-7815.1994
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