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Protein kinase C depresses cardiac myocyte power output and attenuates myofilament responses induced by protein kinase A
Following activation by G-protein-coupled receptor agonists, protein kinase C (PKC) modulates cardiac myocyte function by phosphorylation of intracellular targets including myofilament proteins cardiac troponin I (cTnI) and cardiac myosin binding protein C (cMyBP-C). Since PKC phosphorylation has be...
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| Autors principals: | , , , , , , |
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| Format: | Artigo |
| Idioma: | Inglês |
| Publicat: |
2012
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| Matèries: | |
| Accés en línia: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3568763/ https://ncbi.nlm.nih.gov/pubmed/22527640 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1007/s10974-012-9294-9 |
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