Gamma-amino butyric acid (GABA) prevents the induction of nicotinic receptor-regulated signaling by chronic ethanol in pancreatic cancer cells and normal duct epithelia

Lignende værker

• Effects of chronic nicotine on the autocrine regulation of pancreatic cancer cells and pancreatic duct epithelial cells by stimulatory and inhibitory neurotransmitters
  af: Al-Wadei, Mohammed H., et al.
  Udgivet: (2012)
• Pancreatic cancer cells and normal pancreatic duct epithelial cells express an autocrine catecholamine loop that is activated by nicotinic acetylcholine receptors α3, α5 and α7
  af: Al-Wadei, Mohammed H, et al.
  Udgivet: (2011)
• Differential modulation of nicotine-induced gemcitabine resistance by GABA receptor agonists in pancreatic cancer cell xenografts and in vitro
  af: Banerjee, Jheelam, et al.
  Udgivet: (2014)
• Nicotine induces self-renewal of pancreatic cancer stem cells via neurotransmitter-driven activation of sonic hedgehog signaling
  af: Al-Wadei, Mohammed H., et al.
  Udgivet: (2015)
• Celecoxib and GABA cooperatively prevent the progression of pancreatic cancer in vitro and in xenograft models of stress-free and stress-exposed mice.
  af: Hussein A N Al-Wadei, et al.
  Udgivet: (2012-01-01)