Effects of chronic nicotine on the autocrine regulation of pancreatic cancer cells and pancreatic duct epithelial cells by stimulatory and inhibitory neurotransmitters

Podobne zapisy

• Pancreatic cancer cells and normal pancreatic duct epithelial cells express an autocrine catecholamine loop that is activated by nicotinic acetylcholine receptors α3, α5 and α7
  od: Al-Wadei, Mohammed H, i wsp.
  Wydane: (2011)
• Nicotinic receptor-associated modulation of stimulatory and inhibitory neurotransmitters in NNK-induced adenocarcinoma of the lungs and pancreas
  od: Al-Wadei, Hussein A. N., i wsp.
  Wydane: (2009)
• Nicotine stimulates pancreatic cancer xenografts by systemic increase in stress neurotransmitters and suppression of the inhibitory neurotransmitter γ-aminobutyric acid
  od: Al-Wadei, Hussein A. N., i wsp.
  Wydane: (2009)
• Nicotine induces self-renewal of pancreatic cancer stem cells via neurotransmitter-driven activation of sonic hedgehog signaling
  od: Al-Wadei, Mohammed H., i wsp.
  Wydane: (2015)
• Gamma-amino butyric acid (GABA) prevents the induction of nicotinic receptor-regulated signaling by chronic ethanol in pancreatic cancer cells and normal duct epithelia
  od: Al-Wadei, Mohammed H., i wsp.
  Wydane: (2012)