Blockade of NOX2 and STIM1 signaling limits lipopolysaccharide-induced vascular inflammation

During sepsis, acute lung injury (ALI) results from activation of innate immune cells and endothelial cells by endotoxins, leading to systemic inflammation through proinflammatory cytokine overproduction, oxidative stress, and intracellular Ca(2+) overload. Despite considerable investigation, the un...

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Autors principals: Gandhirajan, Rajesh Kumar, Meng, Shu, Chandramoorthy, Harish C., Mallilankaraman, Karthik, Mancarella, Salvatore, Gao, Hui, Razmpour, Roshanak, Yang, Xiao-Feng, Houser, Steven R., Chen, Ju, Koch, Walter J., Wang, Hong, Soboloff, Jonathan, Gill, Donald L., Madesh, Muniswamy
Format: Artigo
Idioma:Inglês
Publicat: American Society for Clinical Investigation 2013
Matèries:
Research Article
Accés en línia:https://ncbi.nlm.nih.gov/pmc/articles/PMC3561818/
https://ncbi.nlm.nih.gov/pubmed/23348743
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1172/JCI65647
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