Blockade of NOX2 and STIM1 signaling limits lipopolysaccharide-induced vascular inflammation

During sepsis, acute lung injury (ALI) results from activation of innate immune cells and endothelial cells by endotoxins, leading to systemic inflammation through proinflammatory cytokine overproduction, oxidative stress, and intracellular Ca(2+) overload. Despite considerable investigation, the un...

وصف كامل

محفوظ في:
التفاصيل البيبلوغرافية
المؤلفون الرئيسيون: Gandhirajan, Rajesh Kumar, Meng, Shu, Chandramoorthy, Harish C., Mallilankaraman, Karthik, Mancarella, Salvatore, Gao, Hui, Razmpour, Roshanak, Yang, Xiao-Feng, Houser, Steven R., Chen, Ju, Koch, Walter J., Wang, Hong, Soboloff, Jonathan, Gill, Donald L., Madesh, Muniswamy
التنسيق: Artigo
اللغة:Inglês
منشور في: American Society for Clinical Investigation 2013
الموضوعات:
Research Article
الوصول للمادة أونلاين:https://ncbi.nlm.nih.gov/pmc/articles/PMC3561818/
https://ncbi.nlm.nih.gov/pubmed/23348743
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1172/JCI65647
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