ROS-induced ATF3 causes susceptibility to secondary infections during sepsis-associated immunosuppression

Sepsis, sepsis-induced hyperinflammation and subsequent sepsis-associated immunosuppression (SAIS) are important causes of death. Here we show in humans that the loss of the major reactive oxygen species (ROS) scavenger, glutathione (GSH), during SAIS directly correlates with an increase in the expr...

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Bibliografische gegevens
Hoofdauteurs: Hoetzenecker, Wolfram, Echtenacher, Bernd, Guenova, Emmanuella, Hoetzenecker, Konrad, Woelbing, Florian, Brück, Jürgen, Teske, Anna, Valtcheva, Nadejda, Fuchs, Kerstin, Kneilling, Manfred, Park, Ji-Hyeon, Kim, Kyu-Han, Kim, Kyu-Won, Hoffmann, Petra, Krenn, Claus, Hai, Tsonwin, Ghoreschi, Kamran, Biedermann, Tilo, Röcken, Martin
Formaat: Artigo
Taal:Inglês
Gepubliceerd in: 2011
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Online toegang:https://ncbi.nlm.nih.gov/pmc/articles/PMC3555699/
https://ncbi.nlm.nih.gov/pubmed/22179317
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/nm.2557
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