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Ablation of the Kell/Xk complex alters erythrocyte divalent cation homeostasis
XK is a putative transporter of unknown function that is ubiquitously expressed and linked through disulfide bonds to Kell protein, an endothelin-3 (ET-3)-converting enzyme. We generated three knockout (KO) mice that lacked either Xk, Kell or both proteins and characterized erythrocyte cation levels...
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| 主要な著者: | , , , , |
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| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
2012
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3540154/ https://ncbi.nlm.nih.gov/pubmed/23122227 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.bcmd.2012.10.002 |
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