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Ablation of the Kell/Xk complex alters erythrocyte divalent cation homeostasis

XK is a putative transporter of unknown function that is ubiquitously expressed and linked through disulfide bonds to Kell protein, an endothelin-3 (ET-3)-converting enzyme. We generated three knockout (KO) mice that lacked either Xk, Kell or both proteins and characterized erythrocyte cation levels...

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Bibliographic Details
Main Authors: Rivera, Alicia, Kam, Siok Yuen, Ho, Mengfatt, Romero, Jose R., Lee, Soohee
Format: Artigo
Language:Inglês
Published: 2012
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Online Access:https://ncbi.nlm.nih.gov/pmc/articles/PMC3540154/
https://ncbi.nlm.nih.gov/pubmed/23122227
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.bcmd.2012.10.002
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