Regulation of the instantaneous inward rectifier and the delayed outward rectifier potassium channels by Captopril and Angiotensin II via the Phosphoinositide-3 kinase pathway in volume-overload-induced hypertrophied cardiac myocytes

BACKGROUND: Early development of cardiac hypertrophy may be beneficial but sustained hypertrophic activation leads to myocardial dysfunction. Regulation of the repolarizing currents can be modulated by the activation of humoral factors, such as angiotensin II (ANG II) through protein kinases. The ai...

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Main Authors: Alvin, Zikiar, Laurence, Graham G., Coleman, Bernell R., Zhao, Aiqiu, Hajj-Moussa, Majd, Haddad, Georges E.
Formáid: Artigo
Teanga:Inglês
Foilsithe: International Scientific Literature, Inc. 2011
Ábhair:
Basic Research
Rochtain Ar Líne:https://ncbi.nlm.nih.gov/pmc/articles/PMC3539556/
https://ncbi.nlm.nih.gov/pubmed/21709626
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.12659/MSM.881843
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