Regulation of the instantaneous inward rectifier and the delayed outward rectifier potassium channels by Captopril and Angiotensin II via the Phosphoinositide-3 kinase pathway in volume-overload-induced hypertrophied cardiac myocytes

BACKGROUND: Early development of cardiac hypertrophy may be beneficial but sustained hypertrophic activation leads to myocardial dysfunction. Regulation of the repolarizing currents can be modulated by the activation of humoral factors, such as angiotensin II (ANG II) through protein kinases. The ai...

詳細記述

保存先:
書誌詳細
主要な著者: Alvin, Zikiar, Laurence, Graham G., Coleman, Bernell R., Zhao, Aiqiu, Hajj-Moussa, Majd, Haddad, Georges E.
フォーマット: Artigo
言語:Inglês
出版事項: International Scientific Literature, Inc. 2011
主題:
Basic Research
オンライン･アクセス:https://ncbi.nlm.nih.gov/pmc/articles/PMC3539556/
https://ncbi.nlm.nih.gov/pubmed/21709626
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.12659/MSM.881843
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