Regulation of the instantaneous inward rectifier and the delayed outward rectifier potassium channels by Captopril and Angiotensin II via the Phosphoinositide-3 kinase pathway in volume-overload-induced hypertrophied cardiac myocytes

BACKGROUND: Early development of cardiac hypertrophy may be beneficial but sustained hypertrophic activation leads to myocardial dysfunction. Regulation of the repolarizing currents can be modulated by the activation of humoral factors, such as angiotensin II (ANG II) through protein kinases. The ai...

Täydet tiedot

Tallennettuna:
Bibliografiset tiedot
Päätekijät: Alvin, Zikiar, Laurence, Graham G., Coleman, Bernell R., Zhao, Aiqiu, Hajj-Moussa, Majd, Haddad, Georges E.
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: International Scientific Literature, Inc. 2011
Aiheet:
Basic Research
Linkit:https://ncbi.nlm.nih.gov/pmc/articles/PMC3539556/
https://ncbi.nlm.nih.gov/pubmed/21709626
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.12659/MSM.881843
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