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Neuroprotective Effects of Sulforaphane after Contusive Spinal Cord Injury
Traumatic spinal cord injury (SCI) leads to oxidative stress, calcium mobilization, glutamate toxicity, the release of proinflammatory factors, and depletion of reduced glutathione (GSH) at the site of injury. Induction of the Keap1/Nrf2/ARE pathway can alleviate neurotoxicity by protecting against...
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| 主要な著者: | , , , , , , |
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| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
Mary Ann Liebert, Inc.
2012
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3495118/ https://ncbi.nlm.nih.gov/pubmed/22853439 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1089/neu.2012.2474 |
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