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Insulin fails to enhance mTOR phosphorylation, mitochondrial protein synthesis, and ATP production in human skeletal muscle without amino acid replacement

Systemic insulin administration causes hypoaminoacidemia by inhibiting protein degradation, which may in turn inhibit muscle protein synthesis (PS). Insulin enhances muscle mitochondrial PS and ATP production when hypoaminoacidemia is prevented by exogenous amino acid (AA) replacement. We determined...

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Hlavní autoři: Barazzoni, Rocco, Short, Kevin R., Asmann, Yan, Coenen-Schimke, Jill M., Robinson, Matthew M., Sreekumaran Nair, K.
Médium: Artigo
Jazyk:Inglês
Vydáno: American Physiological Society 2012
Témata:
On-line přístup:https://ncbi.nlm.nih.gov/pmc/articles/PMC3492854/
https://ncbi.nlm.nih.gov/pubmed/22967500
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajpendo.00067.2012
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