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Insulin fails to enhance mTOR phosphorylation, mitochondrial protein synthesis, and ATP production in human skeletal muscle without amino acid replacement

Systemic insulin administration causes hypoaminoacidemia by inhibiting protein degradation, which may in turn inhibit muscle protein synthesis (PS). Insulin enhances muscle mitochondrial PS and ATP production when hypoaminoacidemia is prevented by exogenous amino acid (AA) replacement. We determined...

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Autors principals: Barazzoni, Rocco, Short, Kevin R., Asmann, Yan, Coenen-Schimke, Jill M., Robinson, Matthew M., Sreekumaran Nair, K.
Format: Artigo
Idioma:Inglês
Publicat: American Physiological Society 2012
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Accés en línia:https://ncbi.nlm.nih.gov/pmc/articles/PMC3492854/
https://ncbi.nlm.nih.gov/pubmed/22967500
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajpendo.00067.2012
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