eNOS Deficiency Predisposes Podocytes to Injury in Diabetes

Endothelial nitric oxide synthase (eNOS) deficiency may contribute to the pathogenesis of diabetic nephropathy in both experimental models and humans, but the underlying mechanism is not fully understood. Here, we studied two common sequelae of endothelial dysfunction in diabetes: glomerular capilla...

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מידע ביבליוגרפי
Main Authors: Yuen, Darren A., Stead, Bailey E., Zhang, Yanling, White, Kathryn E., Kabir, M. Golam, Thai, Kerri, Advani, Suzanne L., Connelly, Kim A., Takano, Tomoko, Zhu, Lei, Cox, Alison J., Kelly, Darren J., Gibson, Ian W., Takahashi, Takamune, Harris, Raymond C., Advani, Andrew
פורמט: Artigo
שפה:Inglês
יצא לאור: American Society of Nephrology 2012
נושאים:
Basic Research
גישה מקוונת:https://ncbi.nlm.nih.gov/pmc/articles/PMC3482727/
https://ncbi.nlm.nih.gov/pubmed/22997257
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1681/ASN.2011121170
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