eNOS Deficiency Predisposes Podocytes to Injury in Diabetes

Endothelial nitric oxide synthase (eNOS) deficiency may contribute to the pathogenesis of diabetic nephropathy in both experimental models and humans, but the underlying mechanism is not fully understood. Here, we studied two common sequelae of endothelial dysfunction in diabetes: glomerular capilla...

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Dades bibliogràfiques
Autors principals: Yuen, Darren A., Stead, Bailey E., Zhang, Yanling, White, Kathryn E., Kabir, M. Golam, Thai, Kerri, Advani, Suzanne L., Connelly, Kim A., Takano, Tomoko, Zhu, Lei, Cox, Alison J., Kelly, Darren J., Gibson, Ian W., Takahashi, Takamune, Harris, Raymond C., Advani, Andrew
Format: Artigo
Idioma:Inglês
Publicat: American Society of Nephrology 2012
Matèries:
Basic Research
Accés en línia:https://ncbi.nlm.nih.gov/pmc/articles/PMC3482727/
https://ncbi.nlm.nih.gov/pubmed/22997257
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1681/ASN.2011121170
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