eNOS Deficiency Predisposes Podocytes to Injury in Diabetes

Endothelial nitric oxide synthase (eNOS) deficiency may contribute to the pathogenesis of diabetic nephropathy in both experimental models and humans, but the underlying mechanism is not fully understood. Here, we studied two common sequelae of endothelial dysfunction in diabetes: glomerular capilla...

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Detalhes bibliográficos
Main Authors: Yuen, Darren A., Stead, Bailey E., Zhang, Yanling, White, Kathryn E., Kabir, M. Golam, Thai, Kerri, Advani, Suzanne L., Connelly, Kim A., Takano, Tomoko, Zhu, Lei, Cox, Alison J., Kelly, Darren J., Gibson, Ian W., Takahashi, Takamune, Harris, Raymond C., Advani, Andrew
Formato: Artigo
Idioma:Inglês
Publicado em: American Society of Nephrology 2012
Assuntos:
Basic Research
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC3482727/
https://ncbi.nlm.nih.gov/pubmed/22997257
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1681/ASN.2011121170
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