CIKS (Act1 or TRAF3IP2) mediates Angiotensin-II-induced Interleukin-18 Expression, and Nox2-dependent Cardiomyocyte Hypertrophy

Chronic elevation of angiotensin (Ang)-II can lead to myocardial inflammation, hypertrophy and cardiac failure. The adaptor molecule CIKS (connection to IKK and SAPK/JNK) activates the IκB kinase/nuclear factor (NF)-κB and JNK/activator protein (AP)-1 pathways in autoimmune and inflammatory diseases...

Popoln opis

Shranjeno v:
Bibliografske podrobnosti
Main Authors: Valente, Anthony J., Clark, Robert A., Siddesha, Jalahalli M., Siebenlist, Ulrich, Chandrasekar, Bysani
Format: Artigo
Jezik:Inglês
Izdano: 2012
Teme:
Article
Online dostop:https://ncbi.nlm.nih.gov/pmc/articles/PMC3477707/
https://ncbi.nlm.nih.gov/pubmed/22575763
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.yjmcc.2012.04.009
Oznake: Označite
Brez oznak, prvi označite!

Podobne knjige/članki