CIKS (Act1 or TRAF3IP2) mediates Angiotensin-II-induced Interleukin-18 Expression, and Nox2-dependent Cardiomyocyte Hypertrophy

Chronic elevation of angiotensin (Ang)-II can lead to myocardial inflammation, hypertrophy and cardiac failure. The adaptor molecule CIKS (connection to IKK and SAPK/JNK) activates the IκB kinase/nuclear factor (NF)-κB and JNK/activator protein (AP)-1 pathways in autoimmune and inflammatory diseases...

Descrizione completa

Salvato in:
Dettagli Bibliografici
Autori principali: Valente, Anthony J., Clark, Robert A., Siddesha, Jalahalli M., Siebenlist, Ulrich, Chandrasekar, Bysani
Natura: Artigo
Lingua:Inglês
Pubblicazione: 2012
Soggetti:
Article
Accesso online:https://ncbi.nlm.nih.gov/pmc/articles/PMC3477707/
https://ncbi.nlm.nih.gov/pubmed/22575763
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.yjmcc.2012.04.009
Tags: Aggiungi Tag
Nessun Tag, puoi essere il primo ad aggiungerne! !

Documenti analoghi