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Does the ΔF508-CFTR mutation induce a proinflammatory response in human airway epithelial cells?

In the clinical setting, mutations in the cystic fibrosis transmembrane conductance regulator (CFTR) gene enhance the inflammatory response in the lung to Pseudomonas aeruginosa (P. aeruginosa) infection. However, studies on human airway epithelial cells in vitro have produced conflicting results re...

पूर्ण विवरण

में बचाया:
ग्रंथसूची विवरण
मुख्य लेखकों: Hampton, Thomas H., Ballok, Alicia E., Bomberger, Jennifer M., Rutkowski, Melanie R., Barnaby, Roxanna, Coutermarsh, Bonita, Conejo-Garcia, José R., O'Toole, George A., Stanton, Bruce A.
स्वरूप: Artigo
भाषा:Inglês
प्रकाशित: American Physiological Society 2012
विषय:
ऑनलाइन पहुंच:https://ncbi.nlm.nih.gov/pmc/articles/PMC3468482/
https://ncbi.nlm.nih.gov/pubmed/22821996
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajplung.00226.2011
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