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Molecular defect in combined beta-galactosidase and neuraminidase deficiency in man.
In normal human fibroblasts, an enzymically active 85,000-dalton precursor form of beta-galactosidase is processed, via a number of intermediates, into a mature 64,000-dalton form. In addition there is an enzymically inactive 32,000-dalton component and its 54,000-dalton precursor. In fibroblasts fr...
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| Vydáno v: | Proc Natl Acad Sci U S A |
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| Hlavní autoři: | , , , , |
| Médium: | Artigo |
| Jazyk: | Inglês |
| Vydáno: |
National Academy of Sciences
1982
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| Témata: | |
| On-line přístup: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC346709/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/6812049/ https://ncbi.nlm.nih.govhttps://doi.org/10.1073/pnas.79.15.4535 |
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