Antithrombin III Toyama: replacement of arginine-47 by cysteine in hereditary abnormal antithrombin III that lacks heparin-binding ability.

Structural analyses of a hereditary abnormal antithrombin III, antithrombin III Toyama, which has normal progressive antithrombin activity but no heparin cofactor activity, have been carried out to elucidate the molecular abnormality causing recurrent thrombophlebitis of a patient and to identify an...

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Vydáno v:	Proc Natl Acad Sci U S A
Hlavní autoři:	Koide, T, Odani, S, Takahashi, K, Ono, T, Sakuragawa, N
Médium:	Artigo
Jazyk:	Inglês
Vydáno:	National Academy of Sciences 1984
Témata:	Research Article
On-line přístup:	https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC344661/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/6582486/ https://ncbi.nlm.nih.govhttps://doi.org/10.1073/pnas.81.2.289
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