Diastolic Dysfunction and Thin Filament Dysregulation Resulting From Excitation-Contraction Uncoupling in a Mouse Model of Restrictive Cardiomyopathy

Lignende værker

• '(De-)sensitization' vs. 'Uncoupling': what drives cardiomyopathies in the thin filament? Reply
  af: Marston, Steven, et al.
  Udgivet: (2015)
• Excitation-contraction coupling and uncoupling in airway smooth muscle
  af: Rodger, I. W.
  Udgivet: (1985)
• Junctophilins and μ-calpain: partners in excitation–contraction uncoupling
  af: Toral-Ojeda, I, et al.
  Udgivet: (2013)
• Single histidine-substituted cardiac troponin I confers protection from age-related systolic and diastolic dysfunction
  af: Palpant, Nathan J., et al.
  Udgivet: (2008)
• Long Term Ablation of Protein Kinase A (PKA)-mediated Cardiac Troponin I Phosphorylation Leads to Excitation-Contraction Uncoupling and Diastolic Dysfunction in a Knock-in Mouse Model of Hypertrophic Cardiomyopathy
  af: Dweck, David, et al.
  Udgivet: (2014)