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Control of alternative splicing by forskolin through hnRNP K during neuronal differentiation
The molecular basis of cell signal-regulated alternative splicing at the 3′ splice site remains largely unknown. We isolated a protein kinase A-responsive ribonucleic acid (RNA) element from a 3′ splice site of the synaptosomal-associated protein 25 (Snap25) gene for forskolin-inhibited splicing dur...
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| Hauptverfasser: | , , , , |
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| Format: | Artigo |
| Sprache: | Inglês |
| Veröffentlicht: |
Oxford University Press
2012
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| Schlagworte: | |
| Online Zugang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3439897/ https://ncbi.nlm.nih.gov/pubmed/22684629 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1093/nar/gks504 |
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