PPARγ isoforms differentially regulate metabolic networks to mediate mouse prostatic epithelial differentiation

Recent observations indicate prostatic diseases are comorbidities of systemic metabolic dysfunction. These discoveries revealed fundamental questions regarding the nature of prostate metabolism. We previously showed that prostate-specific ablation of PPARγ in mice resulted in tumorigenesis and activ...

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Detalles Bibliográficos
Main Authors: Strand, D W, Jiang, M, Murphy, T A, Yi, Y, Konvinse, K C, Franco, O E, Wang, Y, Young, J D, Hayward, S W
Formato: Artigo
Idioma:Inglês
Publicado: Nature Publishing Group 2012
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Original Article
Acceso en liña:https://ncbi.nlm.nih.gov/pmc/articles/PMC3434663/
https://ncbi.nlm.nih.gov/pubmed/22874998
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/cddis.2012.99
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