PPARγ isoforms differentially regulate metabolic networks to mediate mouse prostatic epithelial differentiation

Recent observations indicate prostatic diseases are comorbidities of systemic metabolic dysfunction. These discoveries revealed fundamental questions regarding the nature of prostate metabolism. We previously showed that prostate-specific ablation of PPARγ in mice resulted in tumorigenesis and activ...

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Detaylı Bibliyografya
Asıl Yazarlar: Strand, D W, Jiang, M, Murphy, T A, Yi, Y, Konvinse, K C, Franco, O E, Wang, Y, Young, J D, Hayward, S W
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: Nature Publishing Group 2012
Konular:
Original Article
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC3434663/
https://ncbi.nlm.nih.gov/pubmed/22874998
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/cddis.2012.99
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