The RON-receptor regulates pancreatic cancer cell migration through phosphorylation-dependent breakdown of the hemidesmosome

The recepteur d’origine nantais (RON) receptor tyrosine kinase is overexpressed and stimulates invasive growth in pancreatic cancer cells, yet the mechanisms that underlie RON-mediated phenotypes remain poorly characterized. To better understand RON function in pancreatic cancer cells, we sought to...

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Bibliographische Detailangaben
Hauptverfasser: Yu, Peter T., Babicky, Michele, Jaquish, Dawn, French, Randy, Marayuma, Karly, Mose, Evangeline, Niessen, Sherry, Hoover, Heather, Shields, David, Cheresh, David, Cravatt, Benjamin F., Lowy, Andrew M.
Format: Artigo
Sprache:Inglês
Veröffentlicht: 2012
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Article
Online Zugang:https://ncbi.nlm.nih.gov/pmc/articles/PMC3424378/
https://ncbi.nlm.nih.gov/pubmed/22275185
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1002/ijc.27447
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