Functional consequences of human airway smooth muscle phenotype plasticity

Eitemau Tebyg

• The laminin β1-competing peptide YIGSR induces a hypercontractile, hypoproliferative airway smooth muscle phenotype in an animal model of allergic asthma
  gan: Dekkers, Bart GJ, et al.
  Cyhoeddwyd: (2010)
• Allergic sensitization enhances the contribution of Rho-kinase to airway smooth muscle contraction
  gan: Schaafsma, Dedmer, et al.
  Cyhoeddwyd: (2004)
• Protein kinase A and the exchange protein directly activated by cAMP (Epac) modulate phenotype plasticity in human airway smooth muscle
  gan: Roscioni, Sara S, et al.
  Cyhoeddwyd: (2011)
• Arginase strongly impairs neuronal nitric oxide-mediated airway smooth muscle relaxation in allergic asthma
  gan: Maarsingh, Harm, et al.
  Cyhoeddwyd: (2006)
• Muscarinic M(3) receptor-dependent regulation of airway smooth muscle contractile phenotype
  gan: Gosens, Reinoud, et al.
  Cyhoeddwyd: (2004)