Engineered interaction between SUR1 and Kir6.2 that enhances ATP sensitivity in K(ATP) channels

Lignende værker

• Engineered Kir6.2 mutations that correct the trafficking defect of K(ATP) channels caused by specific SUR1 mutations
  af: Zhou, Qing, et al.
  Udgivet: (2013)
• ATP activates ATP-sensitive potassium channels composed of mutant sulfonylurea receptor 1 and Kir6.2 with diminished PIP(2) sensitivity
  af: Pratt, Emily B, et al.
  Udgivet: (2011)
• A Kir6.2 Pore Mutation Causes Inactivation of ATP-Sensitive Potassium Channels by Disrupting PIP(2)-Dependent Gating
  af: Bushman, Jeremy D., et al.
  Udgivet: (2013)
• N-terminal transmembrane domain of SUR1 controls gating of Kir6.2 by modulating channel sensitivity to PIP(2)
  af: Pratt, Emily B., et al.
  Udgivet: (2011)
• Stabilization of the Activity of ATP-sensitive Potassium Channels by Ion Pairs Formed between Adjacent Kir6.2 Subunits
  af: Lin, Yu-Wen, et al.
  Udgivet: (2003)